Intracellular Ca2+ Release and Ischemic Axon Injury: The Trojan Horse Is back

Bruce R. Ransom; Angus M. Brown

doi:10.1016/S0896-6273(03)00602-0

Intracellular Ca²⁺ Release and Ischemic Axon Injury: The Trojan Horse Is back

Bruce R. Ransom
, Angus M. Brown

Research output: Journal Publications and Reviews › RGC 21 - Publication in refereed journal › peer-review

12 Citations (Scopus)

Abstract

Ischemic injury of cells in the central nervous system is typically set in motion by influx of extracellular Ca²⁺. In this issue of Neuron, Stys and colleagues propose that ischemic injury in spinal cord axons is partly the result of ryanodine receptor-mediated release of Ca²⁺ from the endoplasmic reticulum (ER), a site of intracellular Ca²⁺ storage.

Original language	English
Pages (from-to)	2-4
Journal	Neuron
Volume	40
Issue number	1
DOIs	https://doi.org/10.1016/S0896-6273(03)00602-0
Publication status	Published - 25 Sept 2003
Externally published	Yes

Access Output

10.1016/S0896-6273(03)00602-0Licence: Elsevier user license

Other Access Options

Check CityUHK Library

Permanent Link

Right click to copy link

Cite this

@article{d6f990158a7741a8b517cf918c5d40a8,

title = "Intracellular Ca2+ Release and Ischemic Axon Injury: The Trojan Horse Is back",

abstract = "Ischemic injury of cells in the central nervous system is typically set in motion by influx of extracellular Ca2+. In this issue of Neuron, Stys and colleagues propose that ischemic injury in spinal cord axons is partly the result of ryanodine receptor-mediated release of Ca2+ from the endoplasmic reticulum (ER), a site of intracellular Ca2+ storage.",

author = "Ransom, \{Bruce R.\} and Brown, \{Angus M.\}",

year = "2003",

month = sep,

day = "25",

doi = "10.1016/S0896-6273(03)00602-0",

language = "English",

volume = "40",

pages = "2--4",

journal = "Neuron",

issn = "0896-6273",

publisher = "Cell Press",

number = "1",

}

TY - JOUR

T1 - Intracellular Ca2+ Release and Ischemic Axon Injury

T2 - The Trojan Horse Is back

AU - Ransom, Bruce R.

AU - Brown, Angus M.

PY - 2003/9/25

Y1 - 2003/9/25

N2 - Ischemic injury of cells in the central nervous system is typically set in motion by influx of extracellular Ca2+. In this issue of Neuron, Stys and colleagues propose that ischemic injury in spinal cord axons is partly the result of ryanodine receptor-mediated release of Ca2+ from the endoplasmic reticulum (ER), a site of intracellular Ca2+ storage.

AB - Ischemic injury of cells in the central nervous system is typically set in motion by influx of extracellular Ca2+. In this issue of Neuron, Stys and colleagues propose that ischemic injury in spinal cord axons is partly the result of ryanodine receptor-mediated release of Ca2+ from the endoplasmic reticulum (ER), a site of intracellular Ca2+ storage.

UR - https://www.scopus.com/pages/publications/0141638472

UR - https://www.scopus.com/record/pubmetrics.uri?eid=2-s2.0-0141638472&origin=recordpage

U2 - 10.1016/S0896-6273(03)00602-0

DO - 10.1016/S0896-6273(03)00602-0

M3 - RGC 21 - Publication in refereed journal

C2 - 14527426

SN - 0896-6273

VL - 40

SP - 2

EP - 4

JO - Neuron

JF - Neuron

IS - 1

ER -