A novel fusion of TPR and ALK in lung adenocarcinoma

Research output: Journal Publications and Reviews (RGC: 21, 22, 62)21_Publication in refereed journalNot applicablepeer-review

46 Scopus Citations
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Author(s)

  • Yoon-La Choi
  • Maruja E. Lira
  • Mineui Hong
  • Ryong N.am Kim
  • So-Jung Choi
  • Ji-Young Song
  • Kinnari Pandy
  • Derrick L. Mann
  • Joshua A. Stahl
  • Heather E. Peckham
  • Joungho Han
  • Mao Mao
  • Jhingook Kim

Detail(s)

Original languageEnglish
Pages (from-to)563-566
Journal / PublicationJournal of thoracic oncology : official publication of the International Association for the Study of Lung Cancer
Volume9
Issue number4
Publication statusPublished - 1 Apr 2014
Externally publishedYes

Abstract

INTRODUCTION: Anaplastic lymphoma kinase (ALK) fusion is the most common mechanism for overexpression and activation in non-small-cell lung carcinoma. Several fusion partners of ALK have been reported, including echinoderm microtubule-associated protein-like 4, TRK-fused gene, kinesin family member 5B, kinesin light chain 1 (KLC1), protein tyrosine phosphatase and nonreceptor type 3, and huntingtin interacting protein 1 (HIP1).

Citation Format(s)

A novel fusion of TPR and ALK in lung adenocarcinoma. / Choi, Yoon-La; Lira, Maruja E.; Hong, Mineui; Kim, Ryong N.am; Choi, So-Jung; Song, Ji-Young; Pandy, Kinnari; Mann, Derrick L.; Stahl, Joshua A.; Peckham, Heather E.; Zheng, Zongli; Han, Joungho; Mao, Mao; Kim, Jhingook.

In: Journal of thoracic oncology : official publication of the International Association for the Study of Lung Cancer, Vol. 9, No. 4, 01.04.2014, p. 563-566.

Research output: Journal Publications and Reviews (RGC: 21, 22, 62)21_Publication in refereed journalNot applicablepeer-review